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The obtained experimental diabetes in laboratory animals equivalent to rats is an efficient model to review oxidative stress due to persistent hyperglycemia, which thereby depresses the endeavor of the antioxidant defense system and induces the era of free radicals de novo. The free radicals thus formed react with all biological elements. The reactions with these aspects of the cell membrane cause lipid peroxidation 26. The liver comprises a high content fabric of antioxidant enzymes inclusive of catalase and superoxide dismutase which could trap these free radicals. Compounds with antioxidant houses might be in a position to reduce the concentration of free radicals. The lipid peroxidation in diabetes results in greater plasma and hepatic levels of MDA and hydroperoxides.